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Academic Journal

Tau Protein in Normal and Alzheimer's Disease Brain: An Update.

Authors : Johnson, Gail V.W.; Hartigan, Judith A.

Source: Journal of Alzheimer's Disease. 1999, Vol. 1 Issue 4/5, p329. 23p.

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Academic Journal

Tau Protein in Normal and Alzheimer's Disease Brain.

Authors : Johnson, Gail V.W.; Jenkins, Scott M.

Source: Journal of Alzheimer's Disease. 1999, Vol. 1 Issue 4/5, p307. 22p.

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Academic Journal

Expression of nicotinic acetylcholine receptor subunits in the cerebral cortex in Alzheimer’s disease: histotopographical correlation with amyloid plaques and hyperphosphorylated-tau protein.

Authors : Wevers, A.¹ (AUTHOR); Monteggia, L.² (AUTHOR); Nowacki, S.¹ (AUTHOR)

Source: European Journal of Neuroscience. Jul99, Vol. 11 Issue 7, p2551-2565. 15p. 2 Charts.

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Academic Journal

REVIEW: tau protein pathology in Alzheimer’s disease and related disorders.

Authors : Tolnay; Probst; Tolnay

Source: Neuropathology & Applied Neurobiology. Jun99, Vol. 25 Issue 3, p171-187. 17p.

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Academic Journal

The prolyl isomerase Pin1 restores the function of Alzheimer-associated phosphorylated tau protein.

Authors : Lu, Pei-Jung; Wulf, Gerburg; Zhen Zhou, Xiao

Source: Nature. 6/24/1999, Vol. 399 Issue 6738, p784. 5p. 11 Color Photographs, 20 Diagrams, 1 Chart, 4 Graphs.

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Academic Journal

A unifying hypothesis of Alzheimer's disease. II. Pathophysiological processes.

Authors : Heininger, Kurt¹

Source: Human Psychopharmacology: Clinical & Experimental. Dec99, Vol. 14 Issue 8, p525-581. 57p.

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Academic Journal

PHF-Tau from Alzheimer Brain is Rapidly Dephosphorylated and Degraded When Injected into Neurons in situ.

Authors : Hall, Garth F.

Source: Journal of Alzheimer's Disease. 1999, Vol. 1 Issue 6, p379. 8p.

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Academic Journal

TOXIC NEURONAL APOPTOSIS AND MODIFICATIONS OF TAU AND APP GENE AND PROTEIN EXPRESSIONS*.

Authors : HUGON, JACQUES^1,2 (AUTHOR); ESCLAIR, FRANÇOISE¹ (AUTHOR); LESORT, MATHEU¹ (AUTHOR)

Source: Drug Metabolism Reviews. Aug99, Vol. 31 Issue 3, p635-647. 13p.

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Academic Journal

Multifocal glial nodules in a case of Duchenne muscular dystrophy with severe mental retardation.

Authors : Itoh, K.; Jinnai, K.; Tada, K.

Source: Neuropathology. Sep99, Vol. 19 Issue 3, p322-327. 6p.

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Academic Journal

Investigations on oxidative stress and therapeutical implications in dementia.

Authors : Durany, N.; Münch, Gerald; Michel, Tanja

Source: European Archives of Psychiatry & Clinical Neuroscience. 1999 Supplement 3, Vol. 249, pS68. 1p.

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فلترة النتائج

Tau Protein in Normal and Alzheimer's Disease Brain: An Update.

Tau Protein in Normal and Alzheimer's Disease Brain.

Expression of nicotinic acetylcholine receptor subunits in the cerebral cortex in Alzheimer’s disease: histotopographical correlation with amyloid plaques and hyperphosphorylated-tau protein.

REVIEW: tau protein pathology in Alzheimer’s disease and related disorders.

The prolyl isomerase Pin1 restores the function of Alzheimer-associated phosphorylated tau protein.

A unifying hypothesis of Alzheimer's disease. II. Pathophysiological processes.

PHF-Tau from Alzheimer Brain is Rapidly Dephosphorylated and Degraded When Injected into Neurons in situ.

TOXIC NEURONAL APOPTOSIS AND MODIFICATIONS OF TAU AND APP GENE AND PROTEIN EXPRESSIONS*.

Multifocal glial nodules in a case of Duchenne muscular dystrophy with severe mental retardation.

Investigations on oxidative stress and therapeutical implications in dementia.

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