Authors : Marek G; Division of Pulmonary, Critical Care, and Sleep Medicine, University of Florida, Gainesville, Florida, United States of America.; Collinsworth A

Subjects: Mutation*; Inclusion Bodies/Inclusion Bodies/Inclusion Bodies/*pathology ; Liver Cirrhosis/Liver Cirrhosis/Liver Cirrhosis/*pathology

Source: PloS one [PLoS One] 2021 Aug 16; Vol. 16 (8), pp. e0256117. Date of Electronic Publication: 2021 Aug 16 (Print Publication: 2021).Publisher: Public Library of Science Country of Publication: United States NLM ID: 101285081 Publication Model: eCollection Cited Medium: Internet

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Combination of the mutations for improving activity of TEV protease in inclusion bodies.

Authors : Hu J; School of Life Science, Anhui Agricultural University, 130, Changjiang West Road, Hefei, 230036, Anhui Province, People's Republic of China.; Chen Y

Subjects: Mutation*; Endopeptidases/Endopeptidases/Endopeptidases/*metabolism ; Inclusion Bodies/Inclusion Bodies/Inclusion Bodies/*enzymology

Source: Bioprocess and biosystems engineering [Bioprocess Biosyst Eng] 2021 Oct; Vol. 44 (10), pp. 2129-2139. Date of Electronic Publication: 2021 May 25.Publisher: Springer-Verlag Country of Publication: Germany NLM ID: 101088505 Publication Model: Print-Electronic Cited Medium: Internet ISSN:

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Familial tauopathy with P364S MAPT mutation: clinical course, neuropathology and ultrastructure of neuronal tau inclusions.

Authors : Štrafela P; Department of Pathology, University Clinical Centre Maribor, Maribor, Slovenia.; Pleško J

Subjects: Mutation*; Brain/Brain/Brain/*pathology ; Inclusion Bodies/Inclusion Bodies/Inclusion Bodies/*pathology

Source: Neuropathology and applied neurobiology [Neuropathol Appl Neurobiol] 2018 Oct; Vol. 44 (6), pp. 550-562. Date of Electronic Publication: 2018 Jan 07.Publisher: Blackwell Scientific Publications Country of Publication: England NLM ID: 7609829 Publication Model: Print-Electronic Cited Medium: Internet

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Intracytoplasmic filamentous inclusions and IGHV rearrangements in a patient with chronic lymphocytic leukemia.

Authors : Rodríguez CM; a Hospital Nacional de Clínicas, Facultad de Ciencias Médicas , Universidad Nacional de Córdoba , Córdoba , Argentina.; b Facultad de Ciencias Químicas , Centro de Investigación en Bioquímica Clínica e Inmunología (CIBICI-CONICET), Universidad Nacional de Córdoba , Córdoba , Argentina.

Subjects: Gene Rearrangement* ; Mutation*; Immunoglobulin Heavy Chains/Immunoglobulin Heavy Chains/Immunoglobulin Heavy Chains/*genetics

Source: Leukemia & lymphoma [Leuk Lymphoma] 2018 May; Vol. 59 (5), pp. 1239-1243. Date of Electronic Publication: 2017 Sep 03.Publisher: Taylor & Francis Country of Publication: United States NLM ID: 9007422 Publication Model: Print-Electronic Cited Medium: Internet ISSN:

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A method of predicting the in vitro fibril formation propensity of Aβ40 mutants based on their inclusion body levels in E. coli.

Authors : Sanagavarapu K; Lund University, Biochemistry and Structural Biology, Chemical Center, Lund, Sweden. .; Nüske E

Subjects: Mutation*; Amyloid/Amyloid/Amyloid/*metabolism ; Amyloid beta-Peptides/Amyloid beta-Peptides/Amyloid beta-Peptides/*genetics

Source: Scientific reports [Sci Rep] 2019 Mar 06; Vol. 9 (1), pp. 3680. Date of Electronic Publication: 2019 Mar 06.Publisher: Nature Publishing Group Country of Publication: England NLM ID: 101563288 Publication Model: Electronic Cited Medium: Internet ISSN:

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Loss of charge mutations in solvent exposed Lys residues of superoxide dismutase 1 do not induce inclusion formation in cultured cell models.

Authors : Crosby K; Department of Neuroscience, Center for Translational Research in Neurodegenerative Disease, University of Florida, Gainesville, Florida, United States of America.; Crown AM

Subjects: Mutation*; Inclusion Bodies/Inclusion Bodies/Inclusion Bodies/*metabolism ; Superoxide Dismutase-1/Superoxide Dismutase-1/Superoxide Dismutase-1/*genetics

Source: PloS one [PLoS One] 2018 Nov 06; Vol. 13 (11), pp. e0206751. Date of Electronic Publication: 2018 Nov 06 (Print Publication: 2018).Publisher: Public Library of Science Country of Publication: United States NLM ID: 101285081 Publication Model: eCollection Cited Medium: Internet

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Subcellular Localization of Matrin 3 Containing Mutations Associated with ALS and Distal Myopathy.

Authors : Gallego-Iradi MC; Department of Neuroscience, Center for Translational Research in Neurodegenerative Disease, McKnight Brain Institute, University of Florida, Gainesville, Florida, United States of America; Santa Fe HealthCare Alzheimer's Disease Research Center, University of Florida, Gainesville, Florida, United States of America.

Subjects: Amyotrophic Lateral Sclerosis*/Amyotrophic Lateral Sclerosis*/Amyotrophic Lateral Sclerosis*/genetics ; Amyotrophic Lateral Sclerosis*/Amyotrophic Lateral Sclerosis*/Amyotrophic Lateral Sclerosis*/metabolism ; Cytoplasm*/Cytoplasm*/Cytoplasm*/genetics

Source: PloS one [PLoS One] 2015 Nov 03; Vol. 10 (11), pp. e0142144. Date of Electronic Publication: 2015 Nov 03 (Print Publication: 2015).Publisher: Public Library of Science Country of Publication: United States NLM ID: 101285081 Publication Model: eCollection Cited Medium: Internet

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Temporal separation of aggregation and ubiquitination during early inclusion formation in transgenic mice carrying the Huntington's disease mutation.

Authors : Gong B; Department of Pharmacology, University of Cambridge, Cambridge, United Kingdom.; Kielar C

Subjects: Mutation* ; Ubiquitination*; Huntington Disease/Huntington Disease/Huntington Disease/*metabolism

Source: PloS one [PLoS One] 2012; Vol. 7 (7), pp. e41450. Date of Electronic Publication: 2012 Jul 24.Publisher: Public Library of Science Country of Publication: United States NLM ID: 101285081 Publication Model: Print-Electronic Cited Medium: Internet

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Cytoplasmic targeting of mutant poly(A)-binding protein nuclear 1 suppresses protein aggregation and toxicity in oculopharyngeal muscular dystrophy.

Authors : Abu-Baker A; Center for Research in Neuroscience, McGill University, and the McGill University Health Center, 1650 Cedar Avenue, Montreal, Quebec, Canada H3G 1A4.; Laganiere S

Subjects: Mutation*; Inclusion Bodies/Inclusion Bodies/Inclusion Bodies/*metabolism ; Muscular Dystrophy, Oculopharyngeal/Muscular Dystrophy, Oculopharyngeal/Muscular Dystrophy, Oculopharyngeal/*metabolism

Source: Traffic (Copenhagen, Denmark) [Traffic] 2005 Sep; Vol. 6 (9), pp. 766-79.Publisher: Wiley Country of Publication: England NLM ID: 100939340 Publication Model: Print Cited Medium: Print ISSN: 1398-9219 (Print)

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Mutant Rab24 GTPase is targeted to nuclear inclusions.

Authors : Maltese WA; Department of Biochemistry and Molecular Biology, Medical College of Ohio, Toledo, OH 43614, USA. ; Soule G

Subjects: Cell Nucleus/Cell Nucleus/Cell Nucleus/*chemistry ; Inclusion Bodies/Inclusion Bodies/Inclusion Bodies/*chemistry ; Mutation/Mutation/Mutation/*genetics

Source: BMC cell biology [BMC Cell Biol] 2002 Sep 25; Vol. 3, pp. 25. Date of Electronic Publication: 2002 Sep 25.Publisher: BioMed Central Country of Publication: England NLM ID: 100966972 Publication Model: Electronic Cited Medium: Internet ISSN: 1471-2121

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