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Publisher : wiley-blackwell

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Microglia Gravitate toward Amyloid Plaques Surrounded by Externalized Phosphatidylserine via TREM2.

Authors : Park, Jong‐Chan^1,2,3 (AUTHOR); Han, Jong Won⁴ (AUTHOR); Lee, Woochan^4,5 (AUTHOR)

Source: Advanced Science. 9/11/2024, Vol. 11 Issue 34, p1-15. 15p.

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Multi‐fluorine labeled indanone derivatives as potential MRI imaging probes for β‐Amyloid plaques.

Authors : Dai, Yaqian¹ (AUTHOR); Fang, Tangni¹ (AUTHOR); Xu, Yuanhong¹ (AUTHOR)

Source: Chemical Biology & Drug Design. Mar2023, Vol. 101 Issue 3, p650-661. 12p.

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Lysosomal LAMP1 immunoreactivity exists in both diffuse and neuritic amyloid plaques in the human hippocampus.

Authors : Hassiotis, Sofia¹; Manavis, Jim²; Blumbergs, Peter C.²

Source: European Journal of Neuroscience. May2018, Vol. 47 Issue 9, p1043-1053. 11p. 16 Color Photographs, 3 Charts, 1 Graph.

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Extensive cortical spongiform changes with cerebellar small amyloid plaques: The clinicopathological case of MV2K+ C subtype in Creutzfeldt- Jakob disease.

Authors : Araki, Kunihiko¹; Nakano, Yuta²; Kobayashi, Atsushi³

Source: Neuropathology. Dec2014, Vol. 34 Issue 6, p541-546. 6p.

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Accumulation of a repulsive axonal guidance molecule RGMa in amyloid plaques: a possible hallmark of regenerative failure in Alzheimer's disease brains.

Authors : Satoh, J.¹; Tabunoki, H.¹; Ishida, T.²

Source: Neuropathology & Applied Neurobiology. Apr2013, Vol. 39 Issue 2, p109-120. 12p. 3 Color Photographs, 2 Graphs.

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A Phenylbenzothiazole Conjugate with the Tricarbonyl fac-[M(I)(CO)3]+ (M = Re, 99Tc, 99mTc) Core for Imaging of β-Amyloid Plaques.

Authors : Sagnou, Marina¹; Tzanopoulou, Stamatia¹; Raptopoulou, Catherine P.²

Source: European Journal of Inorganic Chemistry. Sep2012, Vol. 2012 Issue 27, p4279-4286. 8p.

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Phenylbutyric acid reduces amyloid plaques and rescues cognitive behavior in AD transgenic mice.

Authors : Wiley, Jesse C.¹; Pettan-Brewer, Christina¹; Ladiges, Warren C.¹

Source: Aging Cell. Jun2011, Vol. 10 Issue 3, p418-428. 11p.

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Accumulation of cellular prion protein within dystrophic neurites of amyloid plaques in the Alzheimer's disease brain.

Authors : Takahashi, Reisuke H.^1,2 ; Tobiume, Minoru²; Sato, Yuko²

Source: Neuropathology. Jun2011, Vol. 31 Issue 3, p208-214. 7p. 3 Color Photographs, 1 Black and White Photograph.

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Ultrastructural study of florid plaques in variant Creutzfeldt–Jakob disease: a comparison with amyloid plaques in kuru, sporadic Creutzfeldt–Jakob disease and Gerstmann–Sträussler–Scheinker disease.

Authors : Sikorska, B.¹; Liberski, P. P.¹; Sobów, T.²

Source: Neuropathology & Applied Neurobiology. Feb2009, Vol. 35 Issue 1, p46-59. 14p. 2 Color Photographs, 7 Diagrams, 3 Charts.

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Evidence that neurones accumulating amyloid can undergo lysis to form amyloid plaques in Alzheimer's disease.

Authors : D'Andrea, M R; Nagele, R G¹; Wang, H-Y

Source: Histopathology. Feb2001, Vol. 38 Issue 2, p120-134. 15p. 22 Black and White Photographs, 6 Graphs.

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فلترة النتائج

Microglia Gravitate toward Amyloid Plaques Surrounded by Externalized Phosphatidylserine via TREM2.

Multi‐fluorine labeled indanone derivatives as potential MRI imaging probes for β‐Amyloid plaques.

Lysosomal LAMP1 immunoreactivity exists in both diffuse and neuritic amyloid plaques in the human hippocampus.

Extensive cortical spongiform changes with cerebellar small amyloid plaques: The clinicopathological case of MV2K+ C subtype in Creutzfeldt- Jakob disease.

Accumulation of a repulsive axonal guidance molecule RGMa in amyloid plaques: a possible hallmark of regenerative failure in Alzheimer's disease brains.

A Phenylbenzothiazole Conjugate with the Tricarbonyl fac-[M(I)(CO)3]+ (M = Re, 99Tc, 99mTc) Core for Imaging of β-Amyloid Plaques.

Phenylbutyric acid reduces amyloid plaques and rescues cognitive behavior in AD transgenic mice.

Accumulation of cellular prion protein within dystrophic neurites of amyloid plaques in the Alzheimer's disease brain.

Ultrastructural study of florid plaques in variant Creutzfeldt–Jakob disease: a comparison with amyloid plaques in kuru, sporadic Creutzfeldt–Jakob disease and Gerstmann–Sträussler–Scheinker disease.

Evidence that neurones accumulating amyloid can undergo lysis to form amyloid plaques in Alzheimer's disease.

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