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Academic Journal

Inactivation of TMEM106A promotes lipopolysaccharide-induced inflammation via the MAPK and NF-κB signaling pathways in macrophages.

Authors : Zhang X; Department of Immunology, Peking University School of Basic Medical Sciences, Beijing, China.; NHC Key Laboratory of Medical Immunology, Peking University, Beijing, China.

Subjects: Extracellular Signal-Regulated MAP Kinases/Extracellular Signal-Regulated MAP Kinases/Extracellular Signal-Regulated MAP Kinases/*immunology ; Lipopolysaccharides/Lipopolysaccharides/Lipopolysaccharides/*toxicity ; MAP Kinase Signaling System/MAP Kinase Signaling System/MAP Kinase Signaling System/*immunology

Source: Clinical and experimental immunology [Clin Exp Immunol] 2021 Jan; Vol. 203 (1), pp. 125-136. Date of Electronic Publication: 2020 Oct 23.Publisher: Oxford University Press Country of Publication: England NLM ID: 0057202 Publication Model: Print-Electronic Cited

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Academic Journal

Translation-dependent unwinding of stem-loops by UPF1 licenses Regnase-1 to degrade inflammatory mRNAs.

Authors : Mino T; Department of Medical Chemistry, Graduate School of Medicine, Kyoto University, Kyoto 606-8501, Japan.; Iwai N

Subjects: Macrophages, Peritoneal/Macrophages, Peritoneal/Macrophages, Peritoneal/*immunology ; Nonsense Mediated mRNA Decay/Nonsense Mediated mRNA Decay/Nonsense Mediated mRNA Decay/*immunology ; Protein Serine-Threonine Kinases/Protein Serine-Threonine Kinases/Protein Serine-Threonine Kinases/*genetics

Source: Nucleic acids research [Nucleic Acids Res] 2019 Sep 19; Vol. 47 (16), pp. 8838-8859.Publisher: Oxford University Press Country of Publication: England NLM ID: 0411011 Publication Model: Print Cited Medium:

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Academic Journal

Aryl Hydrocarbon Receptor Activation Synergistically Induces Lipopolysaccharide-Mediated Expression of Proinflammatory Chemokine (c-c motif) Ligand 20.

Authors : Lahoti TS; Department of Veterinary and Biomedical Sciences, Center for Molecular Toxicology and Carcinogenesis, Penn State University, University Park, Pennsylvania 16802.; Boyer JA

Subjects: Basic Helix-Loop-Helix Transcription Factors/Basic Helix-Loop-Helix Transcription Factors/Basic Helix-Loop-Helix Transcription Factors/*metabolism ; Chemokine CCL20/Chemokine CCL20/Chemokine CCL20/*agonists ; Keratinocytes/Keratinocytes/Keratinocytes/*drug effects

Source: Toxicological sciences : an official journal of the Society of Toxicology [Toxicol Sci] 2015 Nov; Vol. 148 (1), pp. 229-40. Date of Electronic Publication: 2015 Aug 10.Publisher: Oxford University Press Country of Publication: United States NLM ID: 9805461 Publication Model: Print-Electronic

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Academic Journal

Regulatory role of PI3K-protein kinase B on the release of interleukin-1β in peritoneal macrophages from the ascites of cirrhotic patients.

Authors : Tapia-Abellán A; Departamento de Bioquímica, Biología Molecular (B) e Inmunología, Facultad de Medicina, Regional Campus of International Excellence 'Campus Mare Nostrum', IMIB-Arrixaca, Universidad de Murcia, Murcia, Spain.; Ruiz-Alcaraz AJ

Subjects: Ascites/Ascites/Ascites/*immunology ; Interleukin-1beta/Interleukin-1beta/Interleukin-1beta/*metabolism ; Liver Cirrhosis/Liver Cirrhosis/Liver Cirrhosis/*immunology

Source: Clinical and experimental immunology [Clin Exp Immunol] 2014 Dec; Vol. 178 (3), pp. 525-36.Publisher: Oxford University Press Country of Publication: England NLM ID: 0057202 Publication Model: Print Cited Medium:

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Academic Journal

Impaired chemotaxis and cell adhesion due to decrease in several cell-surface receptors in cathepsin E-deficient macrophages.

Authors : Tsukuba T; Department of Dental Pharmacology, Graduate School of Biomedical Sciences, Nagasaki University, Nagasaki, Japan. ; Yanagawa M

Subjects: Chemotaxis*/Chemotaxis*/Chemotaxis*/drug effects; Cathepsin E/Cathepsin E/Cathepsin E/*deficiency ; Macrophages, Peritoneal/Macrophages, Peritoneal/Macrophages, Peritoneal/*cytology

Source: Journal of biochemistry [J Biochem] 2009 May; Vol. 145 (5), pp. 565-73. Date of Electronic Publication: 2009 Jan 27.Publisher: Oxford University Press Country of Publication: England NLM ID: 0376600 Publication Model: Print-Electronic Cited

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Academic Journal

Impaired host defence against Mycobacterium avium in mice with chronic granulomatous disease.

Authors : Fujita M; Research Institute for Diseases of the Chest, Kyushu University, Fukuoka, Japan. ; Harada E

Subjects: Immunity, Innate*; Granulomatous Disease, Chronic/Granulomatous Disease, Chronic/Granulomatous Disease, Chronic/*immunology ; Macrophages, Peritoneal/Macrophages, Peritoneal/Macrophages, Peritoneal/*immunology

Source: Clinical and experimental immunology [Clin Exp Immunol] 2010 Jun; Vol. 160 (3), pp. 457-60. Date of Electronic Publication: 2010 Jan 19.Publisher: Oxford University Press Country of Publication: England NLM ID: 0057202 Publication Model: Print-Electronic Cited

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Academic Journal

Oxidative stress and sodium methyldithiocarbamate-induced modulation of the macrophage response to lipopolysaccharide in vivo.

Authors : Pruett SB; Department of Cellular Biology and Anatomy, Louisiana State University Health Sciences Center, Shreveport, Louisiana 71103, USA. ; Cheng B

Subjects: Oxidative Stress*; Cytokines/Cytokines/Cytokines/*metabolism ; Lipopolysaccharides/Lipopolysaccharides/Lipopolysaccharides/*immunology

Source: Toxicological sciences : an official journal of the Society of Toxicology [Toxicol Sci] 2009 Jun; Vol. 109 (2), pp. 237-46. Date of Electronic Publication: 2009 Apr 01.Publisher: Oxford University Press Country of Publication: United States NLM ID: 9805461 Publication Model: Print-Electronic

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Academic Journal

Role of GRP78/BiP degradation and ER stress in deoxynivalenol-induced interleukin-6 upregulation in the macrophage.

Authors : Shi Y; Department of Food Science and Human Nutrition, Michigan State University, East Lansing, Michigan 48824, USA.; Porter K

Subjects: Endoplasmic Reticulum/Endoplasmic Reticulum/Endoplasmic Reticulum/*drug effects ; Heat-Shock Proteins/Heat-Shock Proteins/Heat-Shock Proteins/*metabolism ; Interleukin-6/Interleukin-6/Interleukin-6/*metabolism

Source: Toxicological sciences : an official journal of the Society of Toxicology [Toxicol Sci] 2009 Jun; Vol. 109 (2), pp. 247-55. Date of Electronic Publication: 2009 Mar 31.Publisher: Oxford University Press Country of Publication: United States NLM ID: 9805461 Publication Model: Print-Electronic

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Academic Journal

Roles of tumour necrosis factor-alpha (TNF-alpha), transforming growth factor-beta (TGF-beta), and IL-10 in the modulation of intercellular adhesion molecule-1 (ICAM-1) expression by macrophages during mycobacterial infection.

Authors : Tomioka H; Department of Microbiology and Immunology, and Department of Otorhinolaryngology, Shimane Medical University, Izumo, Japan. ; Shimizu T

Subjects: Intercellular Adhesion Molecule-1/Intercellular Adhesion Molecule-1/Intercellular Adhesion Molecule-1/*biosynthesis ; Interleukin-10/Interleukin-10/Interleukin-10/*physiology ; Macrophages, Peritoneal/Macrophages, Peritoneal/Macrophages, Peritoneal/*immunology

Source: Clinical and experimental immunology [Clin Exp Immunol] 2000 Dec; Vol. 122 (3), pp. 335-42.Publisher: Oxford University Press Country of Publication: England NLM ID: 0057202 Publication Model: Print Cited Medium:

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Academic Journal

Macrophage protein kinase C: its role in modulating membrane microviscosity and superoxide in leishmanial infection.

Authors : Chakraborty P; Biomembrane Division, Indian Institute of Chemical Biology, Raja S.C. Mullick Road, Jadavpur, Calcutta-700032, India.; Ghosh D

Subjects: Leishmania donovani/Leishmania donovani/Leishmania donovani/*pathogenicity ; Macrophages, Peritoneal/Macrophages, Peritoneal/Macrophages, Peritoneal/*metabolism ; Macrophages, Peritoneal/Macrophages, Peritoneal/Macrophages, Peritoneal/*parasitology

Source: Journal of biochemistry [J Biochem] 2000 Feb; Vol. 127 (2), pp. 185-90.Publisher: Oxford University Press Country of Publication: England NLM ID: 0376600 Publication Model: Print Cited Medium:

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نتائج البحث

فلترة النتائج

Inactivation of TMEM106A promotes lipopolysaccharide-induced inflammation via the MAPK and NF-κB signaling pathways in macrophages.

Translation-dependent unwinding of stem-loops by UPF1 licenses Regnase-1 to degrade inflammatory mRNAs.

Aryl Hydrocarbon Receptor Activation Synergistically Induces Lipopolysaccharide-Mediated Expression of Proinflammatory Chemokine (c-c motif) Ligand 20.

Regulatory role of PI3K-protein kinase B on the release of interleukin-1β in peritoneal macrophages from the ascites of cirrhotic patients.

Impaired chemotaxis and cell adhesion due to decrease in several cell-surface receptors in cathepsin E-deficient macrophages.

Impaired host defence against Mycobacterium avium in mice with chronic granulomatous disease.

Oxidative stress and sodium methyldithiocarbamate-induced modulation of the macrophage response to lipopolysaccharide in vivo.

Role of GRP78/BiP degradation and ER stress in deoxynivalenol-induced interleukin-6 upregulation in the macrophage.

Roles of tumour necrosis factor-alpha (TNF-alpha), transforming growth factor-beta (TGF-beta), and IL-10 in the modulation of intercellular adhesion molecule-1 (ICAM-1) expression by macrophages during mycobacterial infection.

Macrophage protein kinase C: its role in modulating membrane microviscosity and superoxide in leishmanial infection.

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