نتائج البحث

Mycobacterium indicus pranii therapy induces tumor regression in MyD88- and TLR2-dependent manner.

• Authors : Kumar P; PDC-I, National Institute of Immunology, Aruna Asaf Ali Marg, New Delhi, 110067, India.; Dept. of Preventive Oncology, Dr. B. R. Ambedkar Cancer Hospital, All India Institute of Medical Sciences, Ansari Nagar, New Delhi, 110029, India.

Subjects: Melanoma, Experimental/Melanoma, Experimental/Melanoma, Experimental/*immunology ; Myeloid Differentiation Factor 88/Myeloid Differentiation Factor 88/Myeloid Differentiation Factor 88/*immunology ; Nontuberculous Mycobacteria/Nontuberculous Mycobacteria/Nontuberculous Mycobacteria/*immunology

• Source: BMC research notes [BMC Res Notes] 2019 Oct 07; Vol. 12 (1), pp. 648. Date of Electronic Publication: 2019 Oct 07.Publisher: Biomed Central Country of Publication: England NLM ID: 101462768 Publication Model: Electronic Cited Medium:

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Anti-inflammatory protein TSG-6 secreted by bone marrow mesenchymal stem cells attenuates neuropathic pain by inhibiting the TLR2/MyD88/NF-κB signaling pathway in spinal microglia.

• Authors : Yang H; Department of Anesthesiology, Shanghai Pulmonary Hospital, Tongji University School of Medicine, 507 Zhengmin Rd, Shanghai, 200433, China.; Wu L

Subjects: Cell Adhesion Molecules/Cell Adhesion Molecules/Cell Adhesion Molecules/*metabolism ; Mesenchymal Stem Cells/Mesenchymal Stem Cells/Mesenchymal Stem Cells/*metabolism ; Microglia/Microglia/Microglia/*metabolism

• Source: Journal of neuroinflammation [J Neuroinflammation] 2020 May 11; Vol. 17 (1), pp. 154. Date of Electronic Publication: 2020 May 11.Publisher: BioMed Central Country of Publication: England NLM ID: 101222974 Publication Model: Electronic Cited Medium:

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Investigation of the relationship between community-acquired respiratory distress syndrome toxin and the high-mobility group box protein 1-toll-like receptors-myeloid differentiation factor 88 signaling pathway in Mycoplasma pneumoniae pneumonia.

• Authors : Fan, Yujie¹ (AUTHOR); Ding, Ying¹ (AUTHOR); Li, Yuqin¹ (AUTHOR)

• Source: Italian Journal of Pediatrics. 5/3/2022, Vol. 48 Issue 1, p1-6. 6p.

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Association of proinflammatory genes expression with serum interleukin 1β and free fatty acids in metabolically healthy and unhealthy abdominally obese individuals: a case-control study.

• Authors : Amiri P; Student Research Committee, Tabriz University of Medical Sciences, Tabriz, Iran.; Immunology Research Center, Tabriz University of Medical Sciences, Tabriz, Iran.

Subjects: Fatty Acids, Nonesterified/Fatty Acids, Nonesterified/Fatty Acids, Nonesterified/*blood ; Interleukin-1beta/Interleukin-1beta/Interleukin-1beta/*blood ; Metabolic Diseases/Metabolic Diseases/Metabolic Diseases/*immunology

• Source: BMC immunology [BMC Immunol] 2019 Jul 04; Vol. 20 (1), pp. 23. Date of Electronic Publication: 2019 Jul 04.Publisher: BioMed Central Country of Publication: England NLM ID: 100966980 Publication Model: Electronic Cited Medium:

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Activation of Toll-like receptors by Burkholderia pseudomallei.

• Authors : West TE; Department of Medicine, University of Washington, Seattle, Washington, USA. ; Ernst RK

Subjects: Burkholderia pseudomallei/Burkholderia pseudomallei/Burkholderia pseudomallei/*metabolism ; Macrophages/Macrophages/Macrophages/*metabolism ; Melioidosis/Melioidosis/Melioidosis/*metabolism

• Source: BMC immunology [BMC Immunol] 2008 Aug 08; Vol. 9, pp. 46. Date of Electronic Publication: 2008 Aug 08.Publisher: BioMed Central Country of Publication: England NLM ID: 100966980 Publication Model: Electronic Cited Medium:

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Streptococcus pneumoniae synergizes with nontypeable Haemophilus influenzae to induce inflammation via upregulating TLR2.

• Authors : Lim JH; Department of Microbiology and Immunology, University of Rochester Medical Center, Rochester, New York, NY 14642, USA. ; Ha U

Subjects: Haemophilus influenzae* ; Streptococcus pneumoniae*; Haemophilus Infections/Haemophilus Infections/Haemophilus Infections/*immunology

• Source: BMC immunology [BMC Immunol] 2008 Jul 29; Vol. 9, pp. 40. Date of Electronic Publication: 2008 Jul 29.Publisher: BioMed Central Country of Publication: England NLM ID: 100966980 Publication Model: Electronic Cited Medium:

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Induction of beta defensin 2 by NTHi requires TLR2 mediated MyD88 and IRAK-TRAF6-p38MAPK signaling pathway in human middle ear epithelial cells.

• Authors : Lee HY; The Gonda Department of Cell and Molecular Biology, House Ear Institute, Los Angeles, CA, USA. ; Takeshita T

Subjects: Ear, Middle/Ear, Middle/Ear, Middle/*cytology ; Epithelial Cells/Epithelial Cells/Epithelial Cells/*immunology ; Haemophilus influenzae/Haemophilus influenzae/Haemophilus influenzae/*immunology

• Source: BMC infectious diseases [BMC Infect Dis] 2008 Jun 25; Vol. 8, pp. 87. Date of Electronic Publication: 2008 Jun 25.Publisher: BioMed Central Country of Publication: England NLM ID: 100968551 Publication Model: Electronic Cited Medium:

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The Toll-Like receptor adaptor TRIF contributes to otitis media pathogenesis and recovery

• Authors : Michelle L. Hernandez; Kwang Pak; Stephen I. Wasserman

Subjects: lcsh:Immunologic diseases. Allergy; Cell signaling; Immunology

• Source: BMC ImmunologyBMC Immunology, Vol 10, Iss 1, p 45 (2009)

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TLR2 and caspase-1 signaling are critical for bacterial containment but not clearance during craniotomy-associated biofilm infection.

• Authors : Aldrich AL; Department of Pathology and Microbiology, University of Nebraska Medical Center, 985900 Nebraska Medical Center, Omaha, NE, 68198, USA.; Present Address: Moffitt Cancer Center, Tampa, FL, 33612, USA.

Subjects: Biofilms/Biofilms/Biofilms/*growth & development ; Caspase 1/Caspase 1/Caspase 1/*deficiency ; Containment of Biohazards/Containment of Biohazards/Containment of Biohazards/*methods

• Source: Journal of neuroinflammation [J Neuroinflammation] 2020 Apr 14; Vol. 17 (1), pp. 114. Date of Electronic Publication: 2020 Apr 14.Publisher: BioMed Central Country of Publication: England NLM ID: 101222974 Publication Model: Electronic Cited Medium:

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Selenium ameliorates Staphylococcus aureus-induced inflammation in bovine mammary epithelial cells by inhibiting activation of TLR2, NF-κB and MAPK signaling pathways.

• Authors : Wang H; College of Veterinary Medicine, Yangzhou University, Yangzhou, 225009, Jiangsu, China.; Jiangsu Co-innovation Center for Prevention and Control of Important Animal Infectious Diseases and Zoonoses, Yangzhou, 225009, Jiangsu, China.

Subjects: MAP Kinase Signaling System/MAP Kinase Signaling System/MAP Kinase Signaling System/*drug effects ; Mastitis, Bovine/Mastitis, Bovine/Mastitis, Bovine/*drug therapy ; NF-kappa B/NF-kappa B/NF-kappa B/*antagonists & inhibitors

• Source: BMC veterinary research [BMC Vet Res] 2018 Jun 20; Vol. 14 (1), pp. 197. Date of Electronic Publication: 2018 Jun 20.Publisher: BioMed Central Country of Publication: England NLM ID: 101249759 Publication Model: Electronic Cited Medium:

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