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Academic Journal

Synthesis of c-di-GMP analogs modified by ganciclovir and biological activity to induce type I interferon.

Authors : Hong Dai¹; Xiaotong Yu¹; Zhu Guan¹

Source: Journal of Chinese Pharmaceutical Sciences. Dec2019, Vol. 28 Issue 12, p825-834. 10p.

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Academic Journal

Type I interferon gene signature test–low and –high patients with systemic lupus erythematosus have distinct gene expression signatures.

Authors : Brohawn, PZ¹ (AUTHOR); Streicher, K¹ (AUTHOR); Higgs, B W¹ (AUTHOR)

Source: Lupus. Nov2019, Vol. 28 Issue 13, p1524-1533. 10p. 2 Diagrams, 2 Charts, 2 Graphs.

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Academic Journal

Baicalin inhibits influenza virus A replication via activation of type I IFN signaling by reducing miR-146a.

Authors : Li, Rui^1,2,3 (AUTHOR); Wang, Lianxin⁴ (AUTHOR)

Source: Molecular Medicine Reports. Dec2019, Vol. 20 Issue 6, p5041-5049. 9p.

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Academic Journal

Type I interferon limits mast cell–mediated anaphylaxis by controlling secretory granule homeostasis.

Authors : Kobayashi, Toshihiko¹ (AUTHOR) ; Shimabukuro-Demoto, Shiho¹ (AUTHOR); Tsutsui, Hidemitsu¹ (AUTHOR)

Source: PLoS Biology. 11/15/2019, Vol. 17 Issue 11, p1-22. 22p. 3 Color Photographs, 3 Black and White Photographs, 1 Graph.

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Academic Journal

P200 family protein IFI204 negatively regulates type I interferon responses by targeting IRF7 in nucleus.

Authors : Cao, Liu¹ (AUTHOR); Ji, Yanxi^1,2 (AUTHOR); Zeng, Lanyi¹ (AUTHOR)

Source: PLoS Pathogens. 10/11/2019, Vol. 15 Issue 10, p1-22. 22p.

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Academic Journal

Type I interferon-dependent CCL4 is induced by a cGAS/STING pathway that bypasses viral inhibition and protects infected tissue, independent of viral burden.

Authors : Parekh, Nikhil J.¹ (AUTHOR); Krouse, Tracy E.¹ (AUTHOR); Reider, Irene E.¹ (AUTHOR)

Source: PLoS Pathogens. 10/11/2019, Vol. 15 Issue 10, p1-25. 25p.

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Academic Journal

Induction of PGRN by influenza virus inhibits the antiviral immune responses through downregulation of type I interferons signaling.

Authors : Wei, Fanhua^1,2 ; Jiang, Zhimin¹; Sun, Honglei¹

Source: PLoS Pathogens. 10/4/2019, Vol. 15 Issue 10, p1-26. 26p.

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Academic Journal

Activation of type I interferon antiviral response in human neural stem cells.

Authors : Lin, Jhao-Yin^1,2 (AUTHOR); Kuo, Rei-Lin^1,3,4 (AUTHOR); Huang, Hsing-I^1,3,4 (AUTHOR)

Source: Stem Cell Research & Therapy. 12/16/2019, Vol. 10 Issue 1, p1-17. 17p.

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Academic Journal

The ETS transcription factor ELF1 regulates a broadly antiviral program distinct from the type I interferon response.

Authors : Seifert, Leon Louis¹ (AUTHOR); Si, Clara² (AUTHOR); Saha, Debjani³ (AUTHOR)

Source: PLoS Pathogens. 11/4/2019, Vol. 15 Issue 11, p1-34. 34p.

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Academic Journal

STAT2 dependent Type I Interferon response promotes dysbiosis and luminal expansion of the enteric pathogen Salmonella Typhimurium.

Authors : Wilson, R. Paul¹; Tursi, Sarah A.¹; Rapsinski, Glenn J.¹

Source: PLoS Pathogens. 4/22/2019, Vol. 15 Issue 4, p1-22. 22p.

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فلترة النتائج

Synthesis of c-di-GMP analogs modified by ganciclovir and biological activity to induce type I interferon.

Type I interferon gene signature test–low and –high patients with systemic lupus erythematosus have distinct gene expression signatures.

Baicalin inhibits influenza virus A replication via activation of type I IFN signaling by reducing miR-146a.

Type I interferon limits mast cell–mediated anaphylaxis by controlling secretory granule homeostasis.

P200 family protein IFI204 negatively regulates type I interferon responses by targeting IRF7 in nucleus.

Type I interferon-dependent CCL4 is induced by a cGAS/STING pathway that bypasses viral inhibition and protects infected tissue, independent of viral burden.

Induction of PGRN by influenza virus inhibits the antiviral immune responses through downregulation of type I interferons signaling.

Activation of type I interferon antiviral response in human neural stem cells.

The ETS transcription factor ELF1 regulates a broadly antiviral program distinct from the type I interferon response.

STAT2 dependent Type I Interferon response promotes dysbiosis and luminal expansion of the enteric pathogen Salmonella Typhimurium.

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