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Academic Journal

Increased levels of UCHL1 are a compensatory response to disrupted ubiquitin homeostasis in spinal muscular atrophy and do not represent a viable therapeutic target.

Authors : Powis, Rachael A.^1,2; Mutsaers, Chantal A.^1,2; Wishart, Thomas. M.^1,3

Source: Neuropathology & Applied Neurobiology. Dec2014, Vol. 40 Issue 7, p873-887. 15p.

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Academic Journal

Analysis of the C9orf72 gene in spinal muscular atrophy patients.

Authors : Alías, Laura^1,2 (AUTHOR); Bernal, Sara¹ (AUTHOR); Barceló, Maria J.^1,2 (AUTHOR)

Source: Amyotrophic Lateral Sclerosis & Frontotemporal Degeneration. Dec2014, Vol. 15 Issue 7/8, p563-568. 6p.

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Academic Journal

Targeting SR Proteins Improves SMN Expression in Spinal Muscular Atrophy Cells.

Authors : Wee, Claribel D.¹; Havens, Mallory A.¹; Jodelka, Francine M.¹

Source: PLoS ONE. Dec2014, Vol. 9 Issue 12, p1-21. 21p.

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Academic Journal

Human Adipose-Derived Mesenchymal Stem Cells as a New Model of Spinal and Bulbar Muscular Atrophy.

Authors : Dossena, Marta¹; Bedini, Gloria¹; Rusmini, Paola²

Source: PLoS ONE. Nov2014, Vol. 9 Issue 11, p1-8. 8p.

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Academic Journal

A Comparison of Three Electrophysiological Methods for the Assessment of Disease Status in a Mild Spinal Muscular Atrophy Mouse Model.

Authors : Li, Jia¹; Geisbush, Tom R.¹; Arnold, William D.²

Source: PLoS ONE. Oct2014, Vol. 9 Issue 10, p1-7. 7p.

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Academic Journal

Sequencing Technology in Molecular Diagnosis of Spinal Muscular Atrophy Caused by SMN1 Deletion.

Authors : MENG Yingtao¹; SHU Jianbo¹

Source: Tianjin Medical Journal. 2014, Vol. 42 Issue 7, p697-700. 4p.

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Academic Journal

The Smn-Independent Beneficial Effects of Trichostatin A on an Intermediate Mouse Model of Spinal Muscular Atrophy.

Authors : Liu, Hong¹; Yazdani, Armin^1,2; Murray, Lyndsay M.^1,2

Source: PLoS ONE. Jul2014, Vol. 9 Issue 7, p1-9. 9p.

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Academic Journal

Gem Depletion: Amyotrophic Lateral Sclerosis and Spinal Muscular Atrophy Crossover.

Authors : Cauchi, Ruben J.¹

Source: CNS Neuroscience & Therapeutics. Jul2014, Vol. 20 Issue 7, p574-581. 8p.

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Academic Journal

The craniosacral progression of muscle development influences the emergence of neuromuscular junction alterations in a severe murine model for spinal muscular atrophy.

Authors : Voigt, Tilman¹; Neve, Anuja²; Schümperli, Daniel²

Source: Neuropathology & Applied Neurobiology. Jun2014, Vol. 40 Issue 4, p416-434. 19p.

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Academic Journal

Adenoviral-mediated mouse model of motor impairment in distal spinal muscular atrophy type V.

Authors : Seo, Ah Jung¹ (AUTHOR); Park, Byung Sun¹ (AUTHOR); Jeong, Na Young² (AUTHOR)

Source: Animal Cells & Systems. Oct2014, Vol. 18 Issue 5, p311-317. 7p.

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فلترة النتائج

Increased levels of UCHL1 are a compensatory response to disrupted ubiquitin homeostasis in spinal muscular atrophy and do not represent a viable therapeutic target.

Analysis of the C9orf72 gene in spinal muscular atrophy patients.

Targeting SR Proteins Improves SMN Expression in Spinal Muscular Atrophy Cells.

Human Adipose-Derived Mesenchymal Stem Cells as a New Model of Spinal and Bulbar Muscular Atrophy.

A Comparison of Three Electrophysiological Methods for the Assessment of Disease Status in a Mild Spinal Muscular Atrophy Mouse Model.

Sequencing Technology in Molecular Diagnosis of Spinal Muscular Atrophy Caused by SMN1 Deletion.

The Smn-Independent Beneficial Effects of Trichostatin A on an Intermediate Mouse Model of Spinal Muscular Atrophy.

Gem Depletion: Amyotrophic Lateral Sclerosis and Spinal Muscular Atrophy Crossover.

The craniosacral progression of muscle development influences the emergence of neuromuscular junction alterations in a severe murine model for spinal muscular atrophy.

Adenoviral-mediated mouse model of motor impairment in distal spinal muscular atrophy type V.

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