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Academic Journal

Overexpression of α-Synuclein following Methamphetamine.

Authors : MAUCELI, GIUSEPPE¹ (AUTHOR); BUSCETI, CARLA I.² (AUTHOR); PELLEGRINI, ANTONIO¹ (AUTHOR)

Source: Annals of the New York Academy of Sciences. 2006, Vol. 1074 Issue 1, p191-197. 7p. 1 Chart.

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Academic Journal

Mutant α-synuclein-induced degeneration is reduced by parkin in a fly model of Parkinson's disease.

Authors : Haywood, Annika F. M.¹; Staveley, Brian E.¹ ; Hilliker, A. J.

Source: Genome. May2006, Vol. 49 Issue 5, p505-510. 6p. 1 Color Photograph, 1 Chart, 1 Graph.

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Academic Journal

Selective loss of dopaminergic neurons and formation of Lewy body-like aggregations in α-synuclein transgenic fly neuronal cultures.

Authors : Park, Soon S.¹ (AUTHOR); Lee, Daewoo¹ (AUTHOR)

Source: European Journal of Neuroscience. Jun2006, Vol. 23 Issue 11, p2908-2914. 7p. 2 Color Photographs, 3 Graphs.

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Academic Journal

α-Synuclein pathology of the spinal and peripheral autonomic nervous system in neurologically unimpaired elderly subjects.

Authors : Bloch, A.¹; Probst, A.¹; Bissig, H.¹

Source: Neuropathology & Applied Neurobiology. Jun2006, Vol. 32 Issue 3, p284-295. 12p. 1 Diagram, 6 Charts.

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Academic Journal

Attenuation of the norepinephrine transporter activity and trafficking via interactions with α-synuclein.

Authors : Wersinger, Christophe¹ (AUTHOR); Jeannotte, Alexis¹ (AUTHOR); Sidhu, Anita¹ (AUTHOR)

Source: European Journal of Neuroscience. Dec2006, Vol. 24 Issue 11, p3141-3152. 12p. 4 Diagrams, 3 Charts.

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Academic Journal

The Neurotoxin, MPP+, Induces Hyperphosphorylation of Tau, in the Presence of α-Synuclein, in SH-SY5Y Neuroblastoma Cells.

Authors : Duka, Tetyana¹; Sidhu, Anita¹

Source: Neurotoxicity Research. 2006, Vol. 10 Issue 1, p1-10. 10p.

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Academic Journal

Elevated levels of oxidized cholesterol metabolites in Lewy body disease brains accelerate α-synuclein fibrilization.

Authors : Bosco, Daryl A¹; Fowler, Douglas M¹; Qinghai Zhang¹

Source: Nature Chemical Biology. May2006, Vol. 2 Issue 5, p249-253. 5p. 1 Black and White Photograph, 1 Diagram, 2 Graphs.

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Academic Journal

Impairment of microtubule-dependent trafficking by overexpression of α-synuclein.

Authors : Lee, He‐Jin^1,2,3 (AUTHOR); Khoshaghideh, Farnaz¹ (AUTHOR); Lee, Stephen¹ (AUTHOR)

Source: European Journal of Neuroscience. Dec2006, Vol. 24 Issue 11, p3153-3162. 10p. 7 Diagrams, 1 Graph.

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Academic Journal

Convergent Roles of α-Synuclein, DA Metabolism, and the Ubiquitin-Proteasome System in Nigrostriatal Toxicity.

Authors : FORNAI, FRANCESCO^1,2 (AUTHOR); LAZZERI, GLORIA¹ (AUTHOR); DI POGGIO, ADOLFO BANDETTINI¹ (AUTHOR)

Source: Annals of the New York Academy of Sciences. 2006, Vol. 1074 Issue 1, p84-89. 6p. 1 Graph.

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Academic Journal

A spectroscopic study of some of the peptidyl radicals formed following hydroxyl radical attack on β-amyloid and α-synuclein.

Authors : Tabner, Brian J.^1,2 (AUTHOR); Turnbull, Stuart² (AUTHOR); King, Jenny E.² (AUTHOR)

Source: Free Radical Research. Jul2006, Vol. 40 Issue 7, p731-739. 9p.

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فلترة النتائج

Overexpression of α-Synuclein following Methamphetamine.

Mutant α-synuclein-induced degeneration is reduced by parkin in a fly model of Parkinson's disease.

Selective loss of dopaminergic neurons and formation of Lewy body-like aggregations in α-synuclein transgenic fly neuronal cultures.

α-Synuclein pathology of the spinal and peripheral autonomic nervous system in neurologically unimpaired elderly subjects.

Attenuation of the norepinephrine transporter activity and trafficking via interactions with α-synuclein.

The Neurotoxin, MPP+, Induces Hyperphosphorylation of Tau, in the Presence of α-Synuclein, in SH-SY5Y Neuroblastoma Cells.

Elevated levels of oxidized cholesterol metabolites in Lewy body disease brains accelerate α-synuclein fibrilization.

Impairment of microtubule-dependent trafficking by overexpression of α-synuclein.

Convergent Roles of α-Synuclein, DA Metabolism, and the Ubiquitin-Proteasome System in Nigrostriatal Toxicity.

A spectroscopic study of some of the peptidyl radicals formed following hydroxyl radical attack on β-amyloid and α-synuclein.

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