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Publication : respiratory research فلترة بواسطة : موضوع

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Academic Journal

Heme-induced lung injury in human precision cut lung slices: a new model for acute lung injury.

Authors : Kewalramani, Namrata^1,2 (AUTHOR) ; Machahua, Carlos^1,2 (AUTHOR); Marti, Thomas Michael^3,4 (AUTHOR)

Source: Respiratory Research. 4/3/2025, Vol. 26 Issue 1, p1-16. 16p.

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Academic Journal

Heme-induced lung injury in human precision cut lung slices: a new model for acute lung injury.

Authors : Kewalramani, Namrata^1,2 (AUTHOR) ; Machahua, Carlos^1,2 (AUTHOR); Marti, Thomas Michael^3,4 (AUTHOR)

Source: Respiratory Research. 4/2/2025, Vol. 26 Issue 1, p1-16. 16p.

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Academic Journal

EIT guided evaluation of regional ventilation distributions in neonatal and pediatric ARDS: a prospective feasibility study.

Authors : Soltész, Leon¹ (AUTHOR); Leyens, Judith^1,2 (AUTHOR); Vogel, Marieke³ (AUTHOR)

Source: Respiratory Research. 2/19/2025, Vol. 26 Issue 1, p1-12. 12p.

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Academic Journal

Effect of prone position on ventilation-perfusion matching in patients with moderate to severe ARDS with different clinical phenotypes.

Authors : Yang, Shuhe¹ (AUTHOR); Sun, Qin¹ (AUTHOR); Yuan, Xueyan¹ (AUTHOR)

Source: Respiratory Research. 2/28/2025, Vol. 26 Issue 1, p1-12. 12p.

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Academic Journal

Gravitational distribution of regional intrapulmonary shunt assessed by EIT in ARDS.

Authors : Xu, Mengru¹ (AUTHOR) ; Chi, Yi¹ (AUTHOR) ; Yuan, Siyi¹ (AUTHOR)

Source: Respiratory Research. 2/22/2025, Vol. 26 Issue 1, p1-12. 12p.

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Academic Journal

Pathophysiological mechanisms of ARDS: a narrative review from molecular to organ-level perspectives.

Authors : Zhou, Kaihuan¹ (AUTHOR); Qin, Qianqian¹ (AUTHOR); Lu, Junyu¹ (AUTHOR)

Source: Respiratory Research. 2/13/2025, Vol. 26 Issue 1, p1-13. 13p.

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Academic Journal

Cortisol regulates neonatal lung development via Smoothened.

Authors : Lu, Shanshan¹ (AUTHOR); Chen, Yifei¹ (AUTHOR); Song, Jiawen² (AUTHOR)

Source: Respiratory Research. 1/18/2025, Vol. 26 Issue 1, p1-17. 17p.

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Academic Journal

Neutrophil elastase inhibitor (Sivelestat) in the treatment of acute respiratory distress syndrome induced by COVID-19: a multicenter retrospective cohort study.

Authors : Li, Yuting¹ (AUTHOR); Zhao, Jianjun² (AUTHOR); Wei, Jiahui² (AUTHOR)

Source: Respiratory Research. 1/18/2025, Vol. 26 Issue 1, p1-10. 10p.

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Academic Journal

Neutrophil elastase inhibitor (Sivelestat) in the treatment of acute respiratory distress syndrome induced by COVID-19: a multicenter retrospective cohort study.

Authors : Li, Yuting¹ (AUTHOR); Zhao, Jianjun² (AUTHOR); Wei, Jiahui² (AUTHOR)

Source: Respiratory Research. 1/19/2025, Vol. 26 Issue 1, p1-10. 10p.

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Academic Journal

Inhibition of IRE1α/XBP1 axis alleviates LPS-induced acute lung injury by suppressing TXNIP/NLRP3 inflammasome activation and ERK/p65 signaling pathway.

Authors : Wang, Sijiao¹ (AUTHOR); Hu, Lijuan¹ (AUTHOR); Fu, Yipeng² (AUTHOR)

Source: Respiratory Research. 11/27/2024, Vol. 25 Issue 1, p1-13. 13p.

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فلترة النتائج

Heme-induced lung injury in human precision cut lung slices: a new model for acute lung injury.

Heme-induced lung injury in human precision cut lung slices: a new model for acute lung injury.

EIT guided evaluation of regional ventilation distributions in neonatal and pediatric ARDS: a prospective feasibility study.

Effect of prone position on ventilation-perfusion matching in patients with moderate to severe ARDS with different clinical phenotypes.

Gravitational distribution of regional intrapulmonary shunt assessed by EIT in ARDS.

Pathophysiological mechanisms of ARDS: a narrative review from molecular to organ-level perspectives.

Cortisol regulates neonatal lung development via Smoothened.

Neutrophil elastase inhibitor (Sivelestat) in the treatment of acute respiratory distress syndrome induced by COVID-19: a multicenter retrospective cohort study.

Neutrophil elastase inhibitor (Sivelestat) in the treatment of acute respiratory distress syndrome induced by COVID-19: a multicenter retrospective cohort study.

Inhibition of IRE1α/XBP1 axis alleviates LPS-induced acute lung injury by suppressing TXNIP/NLRP3 inflammasome activation and ERK/p65 signaling pathway.

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