نتائج البحث

Specific interactions between Smad proteins and AP-1 components determine TGFβ-induced breast cancer cell invasion.

• Authors : Sundqvist, A¹; Zieba, A²; Vasilaki, E¹

• Source: Oncogene. 8/1/2013, Vol. 32 Issue 31, p3606-3615. 10p. 1 Black and White Photograph, 5 Graphs.

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LMO4 can interact with Smad proteins and modulate transforming growth factor-β signaling in epithelial cells.

• Authors : Lu, Z.^1,2; Lam, K. S.^1,2; Wang, N.^1,2

• Source: Oncogene. 5/11/2006, Vol. 25 Issue 20, p2920-2930. 11p. 3 Diagrams, 4 Graphs.

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FLRG, an activin-binding protein, is a new target of TGFβ transcription activation through Smad proteins.

• Authors : Bartholin, Laurent; Maguer-Satta, Véronique; Hayette, Sandrine

• Source: Oncogene. 9/6/2001, Vol. 20 Issue 39, p5409. 11p.

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Transcription activation of FLRG and follistatin by activin A, through Smad proteins, participates in a negative feedback loop to modulate activin A function.

• Authors : Bartholin L; INSERM U453, Centre Léon Bérard, 69373 Lyon, France.; Maguer-Satta V

Subjects: Activins/Activins/Activins/*genetics ; Activins/Activins/Activins/*metabolism ; Activins/Activins/Activins/*pharmacology

• Source: Oncogene [Oncogene] 2002 Mar 28; Vol. 21 (14), pp. 2227-35.Publisher: Nature Publishing Group Country of Publication: England NLM ID: 8711562 Publication Model: Print Cited Medium: Print ISSN: 0950-9232

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LMO4 can interact with Smad proteins and modulate transforming growth factor-β signaling in epithelial cells

• Authors : Xiaoling Xu; Ning Wang; Bogi Andersen

Subjects: Cancer Research; TGF alpha; Apoptosis

• Source: Oncogene. 25:2920-2930

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Genome-wide mechanisms of Smad binding.

• Authors : Morikawa, M¹; Koinuma, D²; Miyazono, K¹

• Source: Oncogene. 3/28/2013, Vol. 32 Issue 13, p1609-1615. 7p.

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SMAD4 exerts a tumor-promoting role in hepatocellular carcinoma.

• Authors : Hernanda, P Y; Chen, K; Das, A M¹

• Source: Oncogene. 9/24/2015, Vol. 34 Issue 39, p5055-5068. 14p. 3 Color Photographs, 1 Black and White Photograph, 5 Graphs.

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MicroRNA-885-3p inhibits the growth of HT-29 colon cancer cell xenografts by disrupting angiogenesis via targeting BMPR1A and blocking BMP/Smad/Id1 signaling.

• Authors : Xiao F; Glycochemistry and Glycobiology Laboratory, Shanghai Institute of Materia Medica, Chinese Academy of Sciences, Shanghai, China.; Qiu H

Subjects: Bone Morphogenetic Protein Receptors, Type I/Bone Morphogenetic Protein Receptors, Type I/Bone Morphogenetic Protein Receptors, Type I/*metabolism ; Colonic Neoplasms/Colonic Neoplasms/Colonic Neoplasms/*blood supply ; Inhibitor of Differentiation Protein 1/Inhibitor of Differentiation Protein 1/Inhibitor of Differentiation Protein 1/*antagonists & inhibitors

• Source: Oncogene [Oncogene] 2015 Apr 09; Vol. 34 (15), pp. 1968-78. Date of Electronic Publication: 2014 Jun 02.Publisher: Nature Publishing Group Country of Publication: England NLM ID: 8711562 Publication Model: Print-Electronic Cited Medium: Internet ISSN:

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SARA and RNF11 interact with each other and ESCRT-0 core proteins and regulate degradative EGFR trafficking.

• Authors : Kostaras, E¹; Sflomos, G¹; Pedersen, N M²

• Source: Oncogene. 10/31/2013, Vol. 32 Issue 44, p5220-5232. 13p. 3 Color Photographs, 4 Diagrams, 1 Graph.

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Smad3 regulates E-cadherin via miRNA-200 pathway.

• Authors : Ahn, S-M¹; Cha, J-Y²; Kim, J²

• Source: Oncogene. 6/21/2012, Vol. 31 Issue 25, p3051-3059. 9p.

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