نتائج البحث

NF-κB p65-dependent transcriptional regulation of histone deacetylase 2 contributes to the chronic constriction injury-induced neuropathic pain via the microRNA-183/TXNIP/NLRP3 axis.

• Authors : Miao, Jiamin¹ (AUTHOR) ; Zhou, Xuelong² (AUTHOR); Ji, Tianjiao³ (AUTHOR)

• Source: Journal of Neuroinflammation. 7/28/2020, Vol. 17 Issue 1, p1-13. 13p.

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The histone deacetylase inhibitor, sodium butyrate, exhibits neuroprotective effects for ischemic stroke in middle-aged female rats.

• Authors : Min Jung Park¹; Sohrabji, Farida¹

• Source: Journal of Neuroinflammation. 12/1/2016, Vol. 13, p1-14. 14p.

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Microglia-specific deletion of histone deacetylase 3 promotes inflammation resolution, white matter integrity, and functional recovery in a mouse model of traumatic brain injury

• Authors : Yongfang Zhao; Hongfeng Mu; Yichen Huang

Subjects: Conditional gene knockout; Controlled cortical impact; HDAC3

• Source: Journal of Neuroinflammation, Vol 19, Iss 1, Pp 1-19 (2022)

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Neuroprotection by the histone deacetylase inhibitor trichostatin A in a model of lipopolysaccharide-sensitised neonatal hypoxic-ischaemic brain injury.

• Authors : Fleiss, Bobbi^1,2 ; Nilsson, Marie K. L.³; Blomgren, Klas^4,5

• Source: Journal of Neuroinflammation. 2012, Vol. 9 Issue 1, p70-84. 15p. 7 Color Photographs, 1 Diagram, 1 Chart.

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Deletion of myeloid HDAC3 promotes efferocytosis to ameliorate retinal ischemic injury.

• Authors : Shahror, Rami A.¹ (AUTHOR); Shosha, Esraa^1,2 (AUTHOR); Morris, Carol¹ (AUTHOR)

• Source: Journal of Neuroinflammation. 7/12/2024, Vol. 21 Issue 1, p1-21. 21p.

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The histone deacetylase inhibitor suberoylanilide hydroxamic acid attenuates human astrocyte neurotoxicity induced by interferon-γ.

• Authors : Hashioka, Sadayuki^1,2 ; Klegeris, Andis³; McGeer, Patrick L.¹

• Source: Journal of Neuroinflammation. 2012, Vol. 9 Issue 1, p113-120. 8p. 4 Graphs.

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The potential neuroprotective role of a histone deacetylase inhibitor, sodium butyrate, after neonatal hypoxia-ischemia.

• Authors : Jaworska J; NeuroRepair Department, Mossakowski Medical Research Centre, Polish Academy of Sciences, 5 A. Pawinskiego Street, 02-106, Warsaw, Poland.; Ziemka-Nalecz M

Subjects: Butyric Acid/Butyric Acid/Butyric Acid/*therapeutic use ; Histone Deacetylase Inhibitors/Histone Deacetylase Inhibitors/Histone Deacetylase Inhibitors/*therapeutic use ; Hypoxia-Ischemia, Brain/Hypoxia-Ischemia, Brain/Hypoxia-Ischemia, Brain/*metabolism

• Source: Journal of neuroinflammation [J Neuroinflammation] 2017 Feb 10; Vol. 14 (1), pp. 34. Date of Electronic Publication: 2017 Feb 10.Publisher: BioMed Central Country of Publication: England NLM ID: 101222974 Publication Model: Electronic Cited Medium: Internet ISSN: 1742-2094

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NF-κB p65-dependent transcriptional regulation of histone deacetylase 2 contributes to the chronic constriction injury-induced neuropathic pain via the microRNA-183/TXNIP/NLRP3 axis

• Authors : Gang Chen; Tianjiao Ji; Jiamin Miao

Subjects: 0301 basic medicine; Male; Immunology

• Source: Journal of NeuroinflammationJournal of Neuroinflammation, Vol 17, Iss 1, Pp 1-13 (2020)

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Microglia-specific deletion of histone deacetylase 3 promotes inflammation resolution, white matter integrity, and functional recovery in a mouse model of traumatic brain injury.

• Authors : Zhao, Yongfang; Mu, Hongfeng; Huang, Yichen

Subjects: WHITE matter (Nerve tissue); BRAIN injuries; HISTONE deacetylase

• Source: Journal of Neuroinflammation; 8/6/2022, Vol. 19 Issue 1, p1-19, 19p

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HDAC3 inhibition prevents blood-brain barrier permeability through Nrf2 activation in type 2 diabetes male mice.

• Authors : Zhao Q; Department of Neurology, Affiliated Drum Tower Hospital, Medical School of Nanjing University, 321 Zhongshan Rd, Nanjing, 210008, Jiangsu, China.; Neuroprotection Research Laboratory, Departments of Radiology and Neurology, Massachusetts General Hospital and Harvard Medical School, 149 13th Street, Room 2401, Charlestown, Boston, MA, 02129, USA.

Subjects: Blood-Brain Barrier/Blood-Brain Barrier/Blood-Brain Barrier/*metabolism ; Diabetes Mellitus, Type 2/Diabetes Mellitus, Type 2/Diabetes Mellitus, Type 2/*drug therapy ; Diabetes Mellitus, Type 2/Diabetes Mellitus, Type 2/Diabetes Mellitus, Type 2/*metabolism

• Source: Journal of neuroinflammation [J Neuroinflammation] 2019 May 17; Vol. 16 (1), pp. 103. Date of Electronic Publication: 2019 May 17.Publisher: BioMed Central Country of Publication: England NLM ID: 101222974 Publication Model: Electronic Cited Medium: Internet ISSN: 1742-2094

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