Source: International immunology [Int Immunol] 2011 Feb; Vol. 23 (2), pp. 119-28. Date of Electronic Publication: 2011 Jan 27.Publisher: [Oxford University Press Country of Publication: England NLM ID: 8916182 Publication Model: Print-Electronic Cited Medium: Internet ISSN:

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T-cell responses to neurofilament light protein are part of the normal immune repertoire.

Authors : Huizinga R; Department of Immunobiology, Biomedical Primate Research Centre, Rijswijk, The Netherlands.; Hintzen RQ

Subjects: CD4-Positive T-Lymphocytes/CD4-Positive T-Lymphocytes/CD4-Positive T-Lymphocytes/*immunology ; CD8-Positive T-Lymphocytes/CD8-Positive T-Lymphocytes/CD8-Positive T-Lymphocytes/*immunology ; Multiple Sclerosis/Multiple Sclerosis/Multiple Sclerosis/*immunology

Source: International immunology [Int Immunol] 2009 Apr; Vol. 21 (4), pp. 433-41. Date of Electronic Publication: 2009 Feb 24.Publisher: [Oxford University Press Country of Publication: England NLM ID: 8916182 Publication Model: Print-Electronic Cited Medium: Internet ISSN:

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Anti-thymocyte globulin (ATG) prevents autoimmune encephalomyelitis by expanding myelin antigen-specific Foxp3+ regulatory T cells.

Authors : Chung DT; Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School, 77 Avenue Louis Pasteur, HIM 780, Boston, MA 02115, USA.; Korn T

Subjects: Antibodies/Antibodies/Antibodies/*metabolism ; Antilymphocyte Serum/Antilymphocyte Serum/Antilymphocyte Serum/*immunology ; Encephalomyelitis, Autoimmune, Experimental/Encephalomyelitis, Autoimmune, Experimental/Encephalomyelitis, Autoimmune, Experimental/*immunology

Source: International immunology [Int Immunol] 2007 Aug; Vol. 19 (8), pp. 1003-10. Date of Electronic Publication: 2007 Aug 13.Publisher: [Oxford University Press Country of Publication: England NLM ID: 8916182 Publication Model: Print-Electronic Cited Medium: Print ISSN:

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A BAFF antagonist suppresses experimental autoimmune encephalomyelitis by targeting cell-mediated and humoral immune responses.

Authors : Huntington ND; Neuroimmunology Laboratory, Department of Biochemistry, La Trobe University, Bundoora, Victoria 3086, Australia.; Tomioka R

Subjects: Antibody Formation/Antibody Formation/Antibody Formation/*drug effects ; B-Cell Activating Factor/B-Cell Activating Factor/B-Cell Activating Factor/*antagonists & inhibitors ; B-Cell Maturation Antigen/B-Cell Maturation Antigen/B-Cell Maturation Antigen/*administration & dosage

Source: International immunology [Int Immunol] 2006 Oct; Vol. 18 (10), pp. 1473-85. Date of Electronic Publication: 2006 Aug 16.Publisher: [Oxford University Press Country of Publication: England NLM ID: 8916182 Publication Model: Print-Electronic Cited Medium: Print ISSN:

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Abnormal T cell activation caused by the imbalance of the IL-1/IL-1R antagonist system is responsible for the development of experimental autoimmune encephalomyelitis.

Authors : Matsuki T; Center for Experimental Medicine, Institute of Medical Science, University of Tokyo, Minato-ku, Tokyo 108-8639, Japan.; Nakae S

Subjects: Encephalomyelitis, Autoimmune, Experimental/Encephalomyelitis, Autoimmune, Experimental/Encephalomyelitis, Autoimmune, Experimental/*immunology ; Interleukin-1/Interleukin-1/Interleukin-1/*physiology ; Lymphocyte Activation/Lymphocyte Activation/Lymphocyte Activation/*immunology

Source: International immunology [Int Immunol] 2006 Feb; Vol. 18 (2), pp. 399-407. Date of Electronic Publication: 2006 Jan 13.Publisher: [Oxford University Press Country of Publication: England NLM ID: 8916182 Publication Model: Print-Electronic Cited Medium: Print ISSN:

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Co-infection with Trypanosoma brucei brucei prevents experimental autoimmune encephalomyelitis in DBA/1 mice through induction of suppressor APCs.

Authors : Wållberg M; Applied Immunology Unit, Centre for Molecular Medicine L8:04, Karolinska Institute, SE-17176 Stockholm, Sweden. ; Harris RA

Subjects: Encephalomyelitis, Autoimmune, Experimental/Encephalomyelitis, Autoimmune, Experimental/Encephalomyelitis, Autoimmune, Experimental/*immunology ; Myelin-Associated Glycoprotein/Myelin-Associated Glycoprotein/Myelin-Associated Glycoprotein/*immunology ; Trypanosoma brucei brucei/Trypanosoma brucei brucei/Trypanosoma brucei brucei/*immunology

Source: International immunology [Int Immunol] 2005 Jun; Vol. 17 (6), pp. 721-8. Date of Electronic Publication: 2005 May 17.Publisher: [Oxford University Press Country of Publication: England NLM ID: 8916182 Publication Model: Print-Electronic Cited Medium: Print ISSN:

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Anti-MOG autoantibodies in Italian multiple sclerosis patients: specificity, sensitivity and clinical association.

Authors : Mantegazza R; Immunology and Muscular Pathology - Neurology IV, Istituto Nazionale Neurologico 'Carlo Besta', Milan, Italy. ; Cristaldini P

Subjects: Autoantibodies/Autoantibodies/Autoantibodies/*blood ; Multiple Sclerosis/Multiple Sclerosis/Multiple Sclerosis/*immunology ; Myelin-Associated Glycoprotein/Myelin-Associated Glycoprotein/Myelin-Associated Glycoprotein/*immunology

Source: International immunology [Int Immunol] 2004 Apr; Vol. 16 (4), pp. 559-65.Publisher: [Oxford University Press Country of Publication: England NLM ID: 8916182 Publication Model: Print Cited Medium: Print ISSN: 0953-8178

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نتائج البحث

فلترة النتائج

Increased T-cell immunity against aquaporin-4 and proteolipid protein in neuromyelitis optica.

Regulatory and pro-inflammatory phenotypes of myelin basic protein-autoreactive T cells in multiple sclerosis.

Anti-MOG autoantibodies in Italian multiple sclerosis patients: specificity, sensitivity and clinical association.

Antigen-specific splenic CD4+ and CD8+ regulatory T cells generated via the eye, suppress experimental autoimmune encephalomyelitis either at the priming or at the effector phase.

T-cell responses to neurofilament light protein are part of the normal immune repertoire.

Anti-thymocyte globulin (ATG) prevents autoimmune encephalomyelitis by expanding myelin antigen-specific Foxp3+ regulatory T cells.

A BAFF antagonist suppresses experimental autoimmune encephalomyelitis by targeting cell-mediated and humoral immune responses.

Abnormal T cell activation caused by the imbalance of the IL-1/IL-1R antagonist system is responsible for the development of experimental autoimmune encephalomyelitis.

Co-infection with Trypanosoma brucei brucei prevents experimental autoimmune encephalomyelitis in DBA/1 mice through induction of suppressor APCs.

Anti-MOG autoantibodies in Italian multiple sclerosis patients: specificity, sensitivity and clinical association.

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