Authors : Schreiber A; Institute of Virology Muenster (IVM), Centre for Molecular Biology of Inflammation (ZMBE), Westfälische Wilhelms-Universität, Muenster, Germany.; Liedmann S

Subjects: Influenza B virus*/Influenza B virus*/Influenza B virus*/enzymology ; Influenza B virus*/Influenza B virus*/Influenza B virus*/immunology; Interferon Type I/Interferon Type I/Interferon Type I/*antagonists & inhibitors

Source: Cellular microbiology [Cell Microbiol] 2020 Feb; Vol. 22 (2), pp. e13143. Date of Electronic Publication: 2019 Dec 08.Publisher: Hindawi Country of Publication: India NLM ID: 100883691 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1462-5822

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MicroRNA‐206 inhibits influenza A virus replication by targeting tankyrase 2.

Authors : Bamunuarachchi, Gayan^1,2 (AUTHOR); Yang, Xiaoyun^1,2 (AUTHOR); Huang, Chaoqun^1,2 (AUTHOR)

Subjects: *INFLUENZA A virus; *VIRAL replication; *INTERFERON receptors

Source: Cellular Microbiology. Feb2021, Vol. 23 Issue 2, p1-15. 15p.

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Human microRNA‐30 inhibits influenza virus infection by suppressing the expression of SOCS1, SOCS3, and NEDD4.

Authors : Lin, Xian^1,2,3 (AUTHOR); Yu, Shiman^1,2 (AUTHOR); Ren, Peilei^1,2 (AUTHOR)

Subjects: *TYPE I interferons; *INFLUENZA A virus; *VIRUS diseases

Source: Cellular Microbiology. May2020, Vol. 22 Issue 5, p1-15. 15p.

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ESX-1 exploits type I IFN-signalling to promote a regulatory macrophage phenotype refractory to IFNγ-mediated autophagy and growth restriction of intracellular mycobacteria.

Authors : Lienard, Julia¹; Movert, Elin¹; Valfridsson, Christine¹

Subjects: *MYCOBACTERIA; *INTRACELLULAR pathogens; *AUTOPHAGY

Source: Cellular Microbiology. Oct2016, Vol. 18 Issue 10, p1471-1485. 15p.

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Super-infection with Staphylococcus aureus inhibits influenza virus-induced type I IFN signalling through impaired STAT1-STAT2 dimerization.

Authors : Warnking K; Institute of Molecular Virology (IMV), Center for Molecular Biology of Inflammation (ZMBE), Westfaelische Wilhelms-University Muenster, Von Esmarch-Str. 56, D-48149, Muenster, Germany.; Klemm C

Subjects: Host-Pathogen Interactions* ; Virus Replication*; Interferon Type I/Interferon Type I/Interferon Type I/*metabolism

Source: Cellular microbiology [Cell Microbiol] 2015 Mar; Vol. 17 (3), pp. 303-17. Date of Electronic Publication: 2014 Nov 19.Publisher: Hindawi Country of Publication: India NLM ID: 100883691 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1462-5822

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SiiA and SiiB are novel type I secretion system subunits controlling SPI4-mediated adhesion of S almonella enterica.

Authors : Wille, Thorsten¹; Wagner, Carolin^2,3; Mittelstädt, Wilrun³

Subjects: *BACTERIAL adhesins; *SALMONELLA enterica; *EPITHELIAL cells

Source: Cellular Microbiology. Feb2014, Vol. 16 Issue 2, p161-178. 18p.

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Phosphatidylinositol-3-kinase (PI3K) is activated by influenza virus vRNA via the pathogen pattern receptor Rig-I to promote efficient type I interferon production.

Authors : Hrincius, Eike R.¹; Dierkes, Rüdiger¹; Anhlan, Darisuren¹

Subjects: *PHOSPHOLIPIDS; *INFLUENZA viruses; *RNA

Source: Cellular Microbiology. Dec2011, Vol. 13 Issue 12, p1907-1919. 13p.

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فلترة النتائج

Inhibition of type I interferon induction and signalling by mosquito-borne flaviviruses.

Type I interferon promotes cell-to-cell spread of Listeria monocytogenes.

Binding to type I collagen is essential for the infectivity of Vibrio parahaemolyticus to host cells.

Type I interferon antagonistic properties of influenza B virus polymerase proteins.

MicroRNA‐206 inhibits influenza A virus replication by targeting tankyrase 2.

Human microRNA‐30 inhibits influenza virus infection by suppressing the expression of SOCS1, SOCS3, and NEDD4.

ESX-1 exploits type I IFN-signalling to promote a regulatory macrophage phenotype refractory to IFNγ-mediated autophagy and growth restriction of intracellular mycobacteria.

Super-infection with Staphylococcus aureus inhibits influenza virus-induced type I IFN signalling through impaired STAT1-STAT2 dimerization.

SiiA and SiiB are novel type I secretion system subunits controlling SPI4-mediated adhesion of S almonella enterica.

Phosphatidylinositol-3-kinase (PI3K) is activated by influenza virus vRNA via the pathogen pattern receptor Rig-I to promote efficient type I interferon production.

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