Authors : Gilardini Montani MS; Department of Experimental Medicine, 'Sapienza' University of Rome, Laboratory affiliated to Istituto Pasteur Italia-Fondazione Cenci Bolognetti, Rome, Italy.; Tarquini G

Subjects: Epstein-Barr Virus Infections* ; Herpesvirus 4, Human*; Anti-Inflammatory Agents

Source: Carcinogenesis [Carcinogenesis] 2022 Apr 25; Vol. 43 (3), pp. 277-287.Publisher: Irl Press At Oxford University Press Country of Publication: England NLM ID: 8008055 Publication Model: Print Cited Medium: Internet ISSN:

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Elevated lncRNA-UCA1 upregulates EZH2 to promote inflammatory response in sepsis-induced pneumonia via inhibiting HOXA1.

Authors : Zhang, Xiaoqin¹ (AUTHOR); Tang, Xuemei¹ (AUTHOR); Pan, Lingai¹ (AUTHOR)

Subjects: *INFLAMMATION; *LINCRNA; *PNEUMONIA

Source: Carcinogenesis. Apr2022, Vol. 43 Issue 4, p371-381. 11p.

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Protection from inflammatory bowel disease and colitis-associated carcinogenesis with 4-vinyl-2,6-dimethoxyphenol (canolol) involves suppression of oxidative stress and inflammatory cytokines.

Authors : Fang, Jun^1,2,3; Seki, Takahiro^2,4; Tsukamoto, Tetsuya⁵

Subjects: *INFLAMMATORY bowel diseases; *COLITIS; *COLON cancer

Source: Carcinogenesis. Dec2013, Vol. 34 Issue 12, p2833-2841. 9p.

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Anti-inflammatory chemoprevention attenuates the phenotype in a mouse model of esophageal adenocarcinoma.

Authors : Baumeister, Theresa¹ (AUTHOR); Ingermann, Jonas¹ (AUTHOR); Marcazzan, Sabrina^1,2,3 (AUTHOR)

Subjects: *LABORATORY mice; *BARRETT'S esophagus; *ANIMAL disease models

Source: Carcinogenesis. Aug2021, Vol. 42 Issue 8, p1068-1078. 11p.

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p62/SQSTM1 promotes mitophagy and activates the NRF2-mediated antioxidant and anti-inflammatory response restraining EBV-driven B lymphocyte proliferation.

Authors : Montani, Maria Saveria Gilardini¹ (AUTHOR); Tarquini, Greta¹ (AUTHOR); Santarelli, Roberta¹ (AUTHOR)

Subjects: *B cells; *REACTIVE oxygen species; *CELL transformation

Source: Carcinogenesis. Mar2022, Vol. 43 Issue 3, p277-287. 11p.

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Bisphenol A activates EGFR and ERK promoting proliferation, tumor spheroid formation and resistance to EGFR pathway inhibition in estrogen receptor-negative inflammatory breast cancer cells.

Authors : Sauer SJ; Department of Surgery, Division of Surgical Sciences, Duke University Medical Sciences, Durham, NC 27710, USA.; Tarpley M

Subjects: Benzhydryl Compounds/Benzhydryl Compounds/Benzhydryl Compounds/*toxicity ; Carcinogens, Environmental/Carcinogens, Environmental/Carcinogens, Environmental/*toxicity ; ErbB Receptors/ErbB Receptors/ErbB Receptors/*genetics

Source: Carcinogenesis [Carcinogenesis] 2017 Mar 01; Vol. 38 (3), pp. 252-260.Publisher: Irl Press At Oxford University Press Country of Publication: England NLM ID: 8008055 Publication Model: Print Cited Medium: Internet ISSN:

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نتائج البحث

فلترة النتائج

Association between new plasma inflammatory markers and risk of colorectal neoplasms in individuals over 50 years old.

Identification of a common variant with potential pleiotropic effect on risk of inflammatory bowel disease and colorectal cancer

Bisphenol A activates EGFR and ERK promoting proliferation, tumor spheroid formation and resistance to EGFR pathway inhibition in estrogen receptornegative inflammatory breast cancer cells.

Anti-inflammatory natural product goniothalamin reduces colitis-associated and sporadic colorectal tumorigenesis.

p62/SQSTM1 promotes mitophagy and activates the NRF2-mediated antioxidant and anti-inflammatory response restraining EBV-driven B lymphocyte proliferation.

Elevated lncRNA-UCA1 upregulates EZH2 to promote inflammatory response in sepsis-induced pneumonia via inhibiting HOXA1.

Protection from inflammatory bowel disease and colitis-associated carcinogenesis with 4-vinyl-2,6-dimethoxyphenol (canolol) involves suppression of oxidative stress and inflammatory cytokines.

Anti-inflammatory chemoprevention attenuates the phenotype in a mouse model of esophageal adenocarcinoma.

p62/SQSTM1 promotes mitophagy and activates the NRF2-mediated antioxidant and anti-inflammatory response restraining EBV-driven B lymphocyte proliferation.

Bisphenol A activates EGFR and ERK promoting proliferation, tumor spheroid formation and resistance to EGFR pathway inhibition in estrogen receptor-negative inflammatory breast cancer cells.

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