Authors : Li Q; Laboratory of Pathology, Hebei Medical University, No. 361, Zhongshan Eastern Road, Shijiazhuang, Hebei Province, People's Republic of China.; Department of Dermatology, The Third Hospital of Hebei Medical University, No. 139, Ziqiang Road, Shijiazhuang, Hebei Province, People's Republic of China.

Subjects: Organelle Biogenesis*; Apoptosis/Apoptosis/Apoptosis/*drug effects ; Autophagic Cell Death/Autophagic Cell Death/Autophagic Cell Death/*drug effects

Source: Archives of toxicology [Arch Toxicol] 2019 Apr; Vol. 93 (4), pp. 1141-1155. Date of Electronic Publication: 2019 Mar 22.Publisher: Springer-Verlag Country of Publication: Germany NLM ID: 0417615 Publication Model: Print-Electronic Cited Medium: Internet ISSN:

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Academic Journal

Perfluoroalkyl acid exposure induces protective mitochondrial and endoplasmic reticulum autophagy in lung cells.

Authors : Xin, Yan^1,2; Wan, Bin^1,2 ; Yang, Yu¹

Subjects: *HEPATOTOXICOLOGY; *AUTOPHAGY; *NECROSIS

Source: Archives of Toxicology. Oct2018, Vol. 92 Issue 10, p3131-3147. 17p. 3 Color Photographs, 5 Graphs.

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Academic Journal

Autophagy and acetaminophen-induced hepatotoxicity.

Authors : Shan, Shulin¹; Shen, Zhenyu¹; Song, Fuyong¹

Subjects: *AUTOPHAGY; *HEPATOTOXICOLOGY; *ACETAMINOPHEN

Source: Archives of Toxicology. Jul2018, Vol. 92 Issue 7, p2153-2161. 9p. 1 Diagram.

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Academic Journal

Mitochondrial functional impairment in response to environmental toxins in the cardiorenal metabolic syndrome.

Authors : Jia, Guanghong; Aroor, Annayya; Martinez-Lemus, Luis

Subjects: *METABOLIC syndrome; *TOXINS; *POLLUTANTS

Source: Archives of Toxicology. Feb2015, Vol. 89 Issue 2, p147-153. 7p. 1 Diagram.

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Impaired protein adduct removal following repeat administration of subtoxic doses of acetaminophen enhances liver injury in fed mice.

Authors : Nguyen, Nga T.¹ (AUTHOR); Akakpo, Jephte Y.¹ (AUTHOR); Weemhoff, James L.¹ (AUTHOR)

Subjects: *ACETAMINOPHEN; *LIVER injuries; *AUTOPHAGY

Source: Archives of Toxicology. Apr2021, Vol. 95 Issue 4, p1463-1473. 11p.

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Ochratoxin A causes mitochondrial dysfunction, apoptotic and autophagic cell death and also induces mitochondrial biogenesis in human gastric epithelium cells

Authors : Haitao Shen; Jinfeng Cui; Jie Yang

Subjects: 0301 basic medicine; Programmed cell death; Animal food

Source: Archives of Toxicology. 93:1141-1155

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فلترة النتائج

Comparison of iohexol and iodixanol induced nephrotoxicity, mitochondrial damage and mitophagy in a new contrast-induced acute kidney injury rat model.

Comparison of iohexol and iodixanol induced nephrotoxicity, mitochondrial damage and mitophagy in a new contrast-induced acute kidney injury rat model

Ochratoxin A causes mitochondrial dysfunction, apoptotic and autophagic cell death and also induces mitochondrial biogenesis in human gastric epithelium cells.

Activation of autophagy triggers mitochondrial loss and changes acetylation profile relevant for mechanotransduction in bladder cancer cells.

Ochratoxin A causes mitochondrial dysfunction, apoptotic and autophagic cell death and also induces mitochondrial biogenesis in human gastric epithelium cells.

Perfluoroalkyl acid exposure induces protective mitochondrial and endoplasmic reticulum autophagy in lung cells.

Autophagy and acetaminophen-induced hepatotoxicity.

Mitochondrial functional impairment in response to environmental toxins in the cardiorenal metabolic syndrome.

Impaired protein adduct removal following repeat administration of subtoxic doses of acetaminophen enhances liver injury in fed mice.

Ochratoxin A causes mitochondrial dysfunction, apoptotic and autophagic cell death and also induces mitochondrial biogenesis in human gastric epithelium cells

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