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Collection : University Of Luxembourg

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Academic Journal

Glioblastoma hijacks neuronal mechanisms for brain invasion.

Authors : Venkataramani, Varun; Yang, Yvonne; Schubert, Marc Cicero

Subjects: Animals; Astrocytes/pathology; Brain/pathology

Source: Cell, 185 (16), 2899-2917.e31 (2022-08-04)

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Academic Journal

AN1-type zinc finger protein 3 (ZFAND3) is a transcriptional regulator that drives Glioblastoma invasion.

Authors : Schuster, Anne; KLEIN, Eliane; Neirinckx, Virginie

Subjects: COL6A2 protein; human; Cell Adhesion Molecules

Source: Nature Communications, 11 (1), 6366 (2020-12-11)

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Mitochondrial MDM2 Regulates Respiratory Complex I Activity Independently of p53.

Authors : ARENA, Giuseppe; Cissé, Madi Yann; Pyrdziak, Samuel

Subjects: Adenocarcinoma/genetics/metabolism/pathology; Animals; Carcinoma

Source: Molecular Cell, 69 (4), 594-609.e8 (2018)

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EGFR wild-type amplification and activation promote invasion and development of glioblastoma independent of angiogenesis.

Authors : Talasila, Krishna M; Soentgerath, Anke; Euskirchen, Philipp

Subjects: Antibodies; Monoclonal; Humanized

Source: Acta Neuropathologica, 125 (5), 683-98 (2013-05)

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Academic Journal

Anti-VEGF treatment reduces blood supply and increases tumor cell invasion in glioblastoma.

Authors : Keunen, Olivier; Johansson, Mikael; Oudin, Anaïs

Subjects: Antibodies; Monoclonal; Humanized

Source: Proceedings of the National Academy of Sciences of the United States of America, 108 (9), 3749-54 (2011-03-01)

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iTRAQ-based proteomics profiling reveals increased metabolic activity and cellular cross-talk in angiogenic compared with invasive glioblastoma phenotype.

Authors : Rajcevic, Uros; Petersen, Kjell; Knol, Jaco C

Subjects: Aged; 80 and over; Animals

Source: Molecular and Cellular Proteomics, 8 (11), 2595-612 (2009-11)

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فلترة النتائج

Glioblastoma hijacks neuronal mechanisms for brain invasion.

AN1-type zinc finger protein 3 (ZFAND3) is a transcriptional regulator that drives Glioblastoma invasion.

Mitochondrial MDM2 Regulates Respiratory Complex I Activity Independently of p53.

EGFR wild-type amplification and activation promote invasion and development of glioblastoma independent of angiogenesis.

Anti-VEGF treatment reduces blood supply and increases tumor cell invasion in glioblastoma.

iTRAQ-based proteomics profiling reveals increased metabolic activity and cellular cross-talk in angiogenic compared with invasive glioblastoma phenotype.

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