نبذة مختصرة : Articles correspondant à plusieurs colloques organisés dans le cadre des séminaires du CTPS en 2012. ; National audience ; Fanleaf disease is the most serious grapevine viral disease worldwide. The main pathogens responsible for this disease are the Grapevine fanleaf virus (GFLV) and Arabis mosaic virus (ArMV). Both nepoviruses are naturally and specifically transmitted from vine to vine by two distinct species of ectoparasitic nematodes, X index for GFLV and X. diversicaudatum for ArMV. Besides uprooting the infected plants followed by fallow period of at least 7 years, no practical tools are able to eradicate this disease in the vineyard. In addition, no GFLV and/or ArMV source of resistance have been identified. In order to develop a sustainable resistance against fanleaf disease, INRA has undertaken a research program that aims to combine into a rootstock a resistance towards nematodes and a pathogen-derived resistance. To assess the strength and sustainability of genetic resources and the material produced during this program, the pathogenic potential of different populations of nematodes available in collection has been characterized. Our study shows that all X. index populations seem equivalent to evaluate the resistance of the pathosystem GFLV/X. index. In contrast, our work with the pathosystem ArMV/X. diversicaudatum suggests that the origin of the X. diversicaudatum population affects the rate of multiplication on different hosts and its ability to transmit ArMV. Thus, a survey in ArMV infected vineyards were undertaken to collect a viruliferous X. diversicaudatum population originated from grapevine. ; La maladie du court-noué, virose la plus grave de la vigne est répandue dans la quasi-totalité des vignobles du monde. Les principaux agents responsables de cette maladie sont le Grapevine fanleaf virus (GFLV) et l’Arabis mosaic virus (ArMV). Ces deux népovirus, sont transmis spécifiquement de vigne à vigne par deux espèces distinctes de nématodes ectoparasites, Xiphinema index pour le GFLV et X. ...
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